burn reconstruction
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About burn reconstruction
Sources and Guidelines Referenced
The clinical information in this guide is grounded in peer-reviewed evidence and published practice parameters from major reconstructive burn organizations, including the American Burn Association (ABA Practice Guidelines), the International Society for Burn Injuries (ISBI Practice Guidelines for Burn Care 2016/2018), the British Association of Plastic, Reconstructive and Aesthetic Surgeons (BAPRAS), the American Society of Plastic Surgeons (ASPS Evidence-Based Clinical Practice Guidelines), and seminal prospective and retrospective studies (e.g., Engrav et al., 2007; Orgill et al., 2009; McCauley et al., 2012; Herndon et al., Total Burn Care 5th Edition).
Burn Reconstruction: A Comprehensive Patient Guide
1. Definition and Medical Identity
Burn reconstruction is a specialized reconstructive surgical discipline focused on correcting functional and structural deformities caused by deep burn injuries. Known formally as secondary burn reconstruction, this branch of plastic surgery releases tight scar bands, restores skin elasticity, and replaces destroyed tissue to regain joint movement and natural anatomical form.
Unlike primary burn surgery—which prioritizes acute resuscitation, infection prevention, and initial wound closure— secondary burn reconstruction takes place after the initial injury has completely healed. The overarching goal of burn reconstruction is long-term restoration of function, physical comfort, and aesthetic appearance through advanced tissue manipulation.
2. The Underlying Condition or Need
Severe thermal, chemical, electrical, or radiation injuries destroy the dermis, the deep layer of skin responsible for strength and elasticity. During natural healing, the body replaces normal skin with dynamic, fibrous connective tissue. When collagen fibers align abnormally, they create a thickened, unyielding mass known as a hypertrophic scar (a raised, hypervascular scar within original wound boundaries) or a keloid (a scar growing beyond original wound borders).
As scar tissue matures, specialized cells called myofibroblasts (contractile connective tissue cells) pull the edges of the wound together. When this contraction occurs across dynamic anatomical zones—such as the neck, elbow, wrist, hand, knee, or eyelids—it creates a burn scar contracture (a tight band of shortened tissue restricting movement). Untreated contractures cause permanent joint stiffness, structural bone deformities, muscle atrophy, chronic skin breakdown, intractable pain, and profound functional impairment.
3. How the Treatment Works — Mechanism
Burn reconstruction works by surgically interrupting pathological tissue tension and replacing rigid scar tissue with healthy, compliant tissue. Clinicians release or excise the constricting scar matrix, allowing underlying muscles, tendons, and joints to return to their anatomical position. Once the restriction is relieved, the resulting tissue defect is covered using an appropriate reconstructive method.
At the physiological level, introducing well-vascularized tissue or full-thickness skin restores dermal architecture. The transfer of healthy blood vessels improves oxygenation, promotes normal collagen remodeling, and dampens systemic inflammatory signaling. When tissue expansion is utilized, gradual mechanical stretch stimulates mitosis (cellular division) and neo-vascularization in adjacent healthy skin, creating new skin identical in color, texture, and hair-bearing qualities to the lost tissue.
4. Types and Variations
Burn reconstruction encompasses several distinct surgical protocols selected according to the site, depth, size, and vascular status of the deformity. Surgeons tailor the approach based on the reconstructive ladder, advancing from simpler techniques to complex tissue transfers as required.
- Scar Release and Skin Grafting: The contracture band is incised or excised. The defect is covered using a split-thickness skin graft (STSG, containing epidermis and partial dermis) or a full-thickness skin graft (FTSG, containing epidermis and complete dermis). FTSGs resist secondary contraction better than STSGs and are preferred for flexor surfaces and the face.
- Local Flap Tissue Rearrangement: Nearby healthy skin is reorganized using geometric incisions, such as a Z-plasty (a transposed triangular flap technique) or transposition flap. This redirects scar tension lines and adds linear length without requiring donor tissue from another body region.
- Tissue Expansion: A temporary silicone bladder (tissue expander) is placed beneath uninjured skin near the scar. Over 8 to 16 weeks, saline is gradually injected through a port to stretch the overlying skin. In a second procedure, the expander is removed, the scar is excised, and the expanded skin is rotated to cover the defect.
- Regional and Pedicled Flaps: A block of skin, subcutaneous fat, and sometimes muscle attached to its primary blood vessel is moved from an adjacent area to fill deep defects involving exposed bone, joint capsules, or tendons.
- Microvascular Free Flap Transfer: Tissue along with its supplying artery and vein is completely detached from a donor site (e.g., anterolateral thigh or radial forearm) and transferred to the burn site. Surgeons reconnect these tiny micro-vessels under a microscope to local blood vessels (microvascular anastomosis).
| Reconstructive Technique | Primary Mechanism | Best Anatomical Indications | Main Advantage | Main Limitation |
|---|---|---|---|---|
| Z-Plasty / Local Flaps | Geometric redistribution of tissue tension lines | Linear contractures across joint lines, web spaces, facial lines | Uses adjacent matching tissue; no secondary donor site | Requires flexible, unscarred adjacent skin |
| Full-Thickness Graft (FTSG) | Transfer of epidermis and complete dermis | Eyelids, face, palm of hand, flexion creases | Minimal secondary contraction; superior texture and color | Limited donor site availability; requires rich vascular bed |
| Split-Thickness Graft (STSG) | Transfer of epidermis and partial dermis | Large surface area defects, torso, extensive extremity burn coverage | Abundant donor availability; high initial graft intake rate | Prone to secondary contracture and hyperpigmentation |
| Tissue Expansion | Mechanical induction of skin growth via stretch | Scalp reconstruction (alopecia), neck, face, torso scars | Matches donor skin color, texture, and hair pattern perfectly | Requires two separate operations and multi-week expansions |
| Microvascular Free Flap | Transfer of vascularized tissue complex with micro-vessels | Deep defects exposing bone, tendon, joint; severe multi-layer neck/hand contractures | Brings robust blood supply; fills deep structural voids | High technical complexity; requires specialized microsurgical monitoring |
5. Who the Treatment Is For — Indications
Secondary burn reconstruction is indicated for adult and pediatric patients who exhibit functional, anatomical, or symptomatic complications from mature burn scars. Clinical evaluation focuses on contracture severity, scar stability, and functional loss (ISBI Guidelines, 2016).
- Functional Joint Impairment: Contractures restricting the range of motion of the neck, shoulder, elbow, wrist, hand, hip, knee, or ankle.
- Facial and Periorificial Deformities: Scar distortion causing ectropion (everted eyelid leaving the cornea exposed), microstomia (severely restricted mouth opening), impaired nasal breathing, or distorted ear architecture.
- Symptomatic Hypertrophic Scars: Persistent scar tissue causing refractory itching, chronic pain, or repeated skin breakdown under minor friction.
- Scalp Alopecia: Post-burn hair loss amenable to scar excision and tissue expansion or flap rotation.
- Instability and Ulceration: Chronic, non-healing open wounds within old burn scars, which carry a long-term risk of malignant transformation into Marjolin's ulcer (a form of aggressive squamous cell carcinoma).
6. Who the Treatment Is NOT For — Contraindications
Reconstructive burn surgery requires careful patient selection to avoid surgical failure, graft loss, or poor functional recovery. Contraindications are categorized as absolute or relative.
Absolute Contraindications
- Active Scar Immaturity: Operating on red, raised, warm, dynamic scars (typically within 6–12 months of initial injury) leads to severe recurrent scar formation. Surgery is postponed until the scar matures, turns pale, flattens, and softs, unless an uncompromised airway or corneal exposure demands urgent intervention.
- Uncontrolled Active Infection: Active local skin or systemic infection completely precludes grafting or flap procedures.
- Severe Hemodynamic or Medical Instability: Inability to tolerate prolonged general anesthesia due to advanced organ failure.
Relative Contraindications
- Active Tobacco Use: Nicotine causes peripheral vasoconstriction, drastically increasing microvascular free flap failure, skin flap necrosis, and graft loss rates (ASPS Guidelines). Patients must stop nicotine products 4 to 6 weeks preoperatively.
- Uncontrolled Diabetes Mellitus: Poor glycemic control (HbA1c > 8.0%) impairs microvascular healing and increases wound infection risks.
- Non-Compliance with Therapy: Inability or unwillingness to participate in postoperative splinting and rehabilitation compromises reconstructive gains.
- Severe Unmanaged Peripheral Vascular Disease: Impaired arterial blood inflow restricts donor flap options and recipient bed graft intake.
7. Alternatives and Clinical Comparison
Before undergoing surgical reconstruction, or as adjunctive management, non-surgical conservative strategies are evaluated. Choosing between non-surgical scar management and surgical intervention depends on scar severity, functional deficits, and scar maturity stage.
| Treatment Method | Invasiveness | Primary Mode of Action | Expected Outcome | Clinical Trade-Offs |
|---|---|---|---|---|
| Burn Reconstruction Surgery | Invasive (Surgical) | Surgical removal/release of scar with tissue replacement | Definitive release of contractures; immediate gain in tissue length | Surgical risks, requirement for anesthesia, new donor site scars |
| Pressure Garments & Splinting | Non-invasive | External mechanical compression and continuous positioning stretch | Reduces scar thickness and prevents progressive contracture maturation | Requires 23-hour daily wear for 12–18 months; low compliance rate |
| Laser Scar Therapy (e.g., Fractional CO2) | Minimally Invasive | Microscopic thermal channels induce collagen breakdown and remodeling | Improves scar flexibility, texture, pigment, and mild tightness | Requires multiple sessions (3–6+); limited efficacy for severe deep contractures |
| Intralesional Corticosteroid Injections | Minimally Invasive | Inhibits fibroblast proliferation and collagen synthesis | Flattens raised hypertrophic/keloid scars; relieves pain/itching | Can cause subcutaneous fat atrophy, hypopigmentation, and telangiectasias |
8. Pre-Treatment Phase
The preoperative phase begins with a detailed evaluation by a multidisciplinary burn team comprising a reconstructive surgeon, physical/occupational therapist, and scar specialist. The surgeon assesses scar maturity, skin elasticity, range of motion, vascular status, and baseline donor site options.
Diagnostic workup may include Doppler ultrasonography or CT angiography to map donor and recipient blood vessels if a regional or microvascular flap is planned. Baseline functional measurements (goniometric joint range of motion) are documented to establish post-surgical rehabilitation goals.
Patient preparation involves strict nutritional optimization. High-protein nutritional supplementation is recommended to maintain an anabolic state necessary for tissue integration (ABA Guidelines). Patients must discontinue all anticoagulants, anti-inflammatory medications, and herbal supplements 10 to 14 days prior to surgery, under medical supervision. Smoking cessation is verified via urinary cotinine testing when flap reconstruction is planned.
9. The Procedure — Step-by-Step Clinical Detail
Burn reconstruction is performed in an accredited surgical operating suite under general anesthesia or regional nerve blocks, depending on the complexity and location of the procedure. The clinical sequence follows standard reconstructive principles:
Step 1: Patient Positioning and Donor/Recipient Site Preparation
The patient is positioned to allow concurrent access to both the recipient burn site and donor skin sites. The surgical fields are prepared with antiseptic solutions and draped sterilely. Tourniquets are applied to extremities when appropriate to minimize intraoperative blood loss.
Step 2: Scar Release and Excision
The surgeon incises or excises the scar tissue. Contracted fascial bands are carefully divided, releasing deep tension until normal anatomical structures (muscles, blood vessels, nerves) are identified and full passive joint extension is achieved. Absolute hemostasis (bleeding control) is achieved using precise electrocautery to prevent hematoma formation beneath future grafts or flaps.
Step 3: Tissue Harvesting and Preparation
- For Skin Grafting: A dermatome (a precision medical slicing instrument) harvests a split-thickness graft from a selected donor site (e.g., thigh). If a full-thickness graft is needed, the surgeon hand-excises skin down to the subcutaneous fat (e.g., groin or supraclavicular area) and manually removes residual fat from the graft underside.
- For Flaps: Local skin flaps are incised and elevated. For free tissue transfer, the target tissue block and its vascular pedicle are dissected under microscopic visualization.
Step 4: Reconstruction and Insetting
The harvested skin graft or flap is transferred to the recipient site. Local flaps are rotated or transposed (such as in a Z-plasty) and sutured in place under low mechanical tension. Grafts are trimmed, fitted precisely to the defect edges, and secured using absorbable micro-sutures or surgical staples. Small incisions (pie-crusting) may be placed in non-meshed grafts to allow underlying fluid drainage.
Step 5: Microsurgical Anastomosis (Flap Procedures)
If a free flap is utilized, the donor artery and vein are joined to recipient blood vessels using ultra-fine sutures (size 8-0 to 10-0) under an operating microscope. Vascular blood flow is confirmed using intraoperative Doppler probes.
Step 6: Dressing Application and Splinting
Grafts are immobilized using specialized bolster dressings (a tie-over pressure dressing) or continuous negative pressure wound therapy (NPWT) to ensure direct contact between the graft and underlying vascular bed. Custom rigid splints are applied immediately to keep joints in maximum extended elongation.
10. Immediate Post-Procedure Period
Following surgery, patients are transferred to the Post-Anesthesia Care Unit (PACU) or an intensive burn step-down unit. If microvascular free tissue transfer was performed, hourly flap monitoring is initiated using laser Doppler, surface temperature probes, and capillary refill checks to detect early vascular thrombosis (BAPRAS Standards).
Pain management involves a multimodal analgesia protocol combining intravenous non-opioid analgesics, nerve blocks, and oral opioids for breakthrough discomfort. Surgical limbs are elevated above heart level to reduce postoperative edema.
Strict immobilization of the operated region is enforced for the first 3 to 7 days. This prevents shear forces from disrupting microvascular capillary growth into newly placed skin grafts (a process known as imbibition followed by inosculation). Initial wound check and dressing removal typically occur between postoperative days 4 and 7.
11. Recovery — Short and Long Term
Recovery following burn reconstruction is an intensive, structured process that integrates physical therapy, scar management, and clinical follow-up over many months.
Short-Term Recovery (Weeks 1 to 6)
Graft intake is evaluated during the initial dressing change. Once graft survival is established (typically day 5–7), gentle passive range-of-motion exercises are initiated under therapist supervision. Splints are re-applied between therapy sessions and worn continuously at night. Donor sites for split-thickness grafts generally heal within 10 to 14 days under specialized occlusive dressings.
By weeks 3 to 4, active range-of-motion exercises expand. Patients receive custom-measured, medical-grade compression garments (applying 15–25 mmHg pressure) to suppress hypervascularity and hypertrophic collagen formation in newly healed areas.
Long-Term Recovery (Months 2 to 18)
Scar maturation requires 12 to 18 months. During this frame, patients wear pressure garments 23 hours a day and apply continuous silicone gel sheets over surgical seams. Routine outpatient physical therapy prevents secondary joint tightening. Follow-up visits occur at 1, 3, 6, 12, and 18 months to evaluate joint range, scar pliability, contour alignment, and nerve sensory re-innervation.
12. Risks, Side Effects, and Complications
While modern reconstructive procedures are safe, surgical complications can occur. Complications are divided by severity and frequency.
| Risk Category | Potential Complication | Clinical Severity | Management Approach |
|---|---|---|---|
| Common / Mild | Donor site pain and delayed superficial healing | Mild | Topical wound care, oral analgesics, barrier dressings |
| Common / Mild | Hyperpigmentation or hypopigmentation of graft | Mild to Moderate | Sun protection, cosmetic camouflage, laser therapy |
| Uncommon / Moderate | Partial skin graft loss / partial flap necrosis | Moderate | Targeted wound care, topical antimicrobials, delayed healing |
| Uncommon / Moderate | Hematoma or seroma under graft/flap | Moderate | Aspiration, bed-side evacuation, bolster adjustment |
| Rare / Serious | Microvascular free flap venous/arterial thrombosis | Severe | Urgent surgical re-exploration and thrombectomy (<24 hours) |
| Rare / Serious | Surgical site infection leading to total graft loss | Severe | Intravenous antibiotics, debridement, re-grafting |
| Rare / Serious | Recurrent contracture recurrence | Moderate to Severe | Re-initiation of splinting/therapy; secondary revision surgery |
Detailed Complications and Long-Term Safety
Graft Loss: Occurs primary due to fluid accumulation (hematoma/seroma) beneath the graft, infection, or shear forces destroying fragile capillary connections. Complete graft loss requires surgical re-clearing and repeated skin grafting (Engrav et al., 2007).
Flap Failure: Vascular compromise of a free flap occurs in 2% to 5% of cases (Orgill et al., 2009). Early detection of venous congestion or arterial insufficiency within the first 48 hours offers a window for surgical re-exploration and salvage.
Warning Signs: Patients must seek immediate clinical evaluation if they experience fever above 38.5°C (101.3°F), sudden dark discoloration or coolness of grafted/flap tissue, foul-smelling drainage, or sudden severe pain unrelieved by prescribed medication.
13. Lifestyle and Behavioural Considerations
Optimizing functional and aesthetic results requires active patient engagement in lifestyle modifications before and after surgery.
Pre-Treatment Optimisation
- Smoking Cessation: Abstain from all nicotine products (including e-cigarettes and nicotine patches) for a minimum of 4 weeks pre- and postoperatively.
- Nutritional Loading: Adequate caloric and protein intake (1.5–2.0 grams of protein per kilogram of body weight daily) builds nitrogen balances necessary for rapid collagen synthesis.
Post-Treatment Lifestyle Management
- Strict Sun Protection: Reconstructed skin and donor sites lack normal melanocyte control and burn rapidly when exposed to ultraviolet radiation. Continuous application of broad-spectrum SPF 50+ sunscreen and protective clothing is mandatory for 12–18 months to prevent permanent dark discoloration (hyperpigmentation).
- Pressure Garment Compliance: Compression therapy must be maintained consistently. Omitting pressure garments early in recovery markedly increases the risk of hypertrophic scar re-growth.
- Moisturization and Massage: Reconstructed skin grafts lack normal sweat and oil glands. Daily application of unscented emollients accompanied by cross-friction scar massage breaks down rigid collagen cross-links and improves tissue pliability.
14. How Outcomes Are Measured
Clinical success in burn reconstruction is evaluated using objective biomechanical tools and validated patient-reported outcome measures (PROMs).
- Goniometric Range-of-Motion (ROM): Objective measurement of joint angle movement in degrees compared to uninjured contralateral limbs.
- Vancouver Scar Scale (VSS): A standardized score assessing scar vascularity, height/thickness, pliability, and pigmentation. Lower scores indicate scar normalization.
- Patient-Reported Outcome Measures: Instruments such as the BRIEF-A, Burn Specific Health Scale-Brief (BSHS-B), and SCAR-Q quantify physical comfort, daily function, body image satisfaction, and psychological well-being.
While primary functional contracture release succeeds in over 85–90% of cases (McCauley et al., 2012), growing pediatric patients or individuals with massive burn surface areas may require planned staged secondary touch-up revisions as physical growth occurs.
15. Recent Advances and Current Standard of Care
Burn reconstruction has evolved rapidly over the past decade, incorporating biotechnology, laser therapeutics, and advanced microsurgery into standardized care pathways.
Current best practice combines traditional surgical tissue transfer with dermal regenerative matrices (e.g., acellular dermal matrices). These biopolymer scaffolds act as structural templates that guide normal vascular and cellular ingrowth, producing more compliant skin coverage without relying entirely on thick autologous tissue grafts.
Additionally, early intervention with ultrapulsed fractional CO2 laser therapy has reshaped secondary burn care. Applied either before surgery to soften scar beds or months postoperatively to blend graft margins, fractional lasers create micro-thermal treatment zones that stimulate scar remodeling, improve elasticity, and treat stubborn neuropathic itching without open surgery.
16. Common Myths and Misconceptions
Myth: Burn reconstruction surgery will completely erase all scars and return the skin to its exact pre-injury state.
Reality: Reconstructive surgery replaces tight, restrictive, pathological scar tissue with softer, more functional tissue. While form and movement are significantly restored, permanent surgical seam lines and minor texture differences will remain.
Myth: Reconstruction should be performed as soon as initial acute burn wounds heal.
Reality: Operating on immature, hypervascular scars increases surgical bleeding and triggers recurrent contracture. Surgeons prefer waiting 12 to 18 months for complete scar maturation, except in urgent cases involving airway or eye compromise.
Myth: Skin grafts grow and function exactly like uninjured skin right away.
Reality: Grafted skin initially lacks oil glands, sweat glands, and sensory nerves. It requires long-term daily moisturization, sun protection, and months of gradual nerve re-innervation.
Myth: Once the surgery is finished, the physical repair is complete.
Reality: Surgery is only one phase of reconstruction. Postoperative success relies on months of dedicated physical therapy, custom splinting, and pressure garment compliance.
Myth: Laser scar treatments can replace the need for reconstructive surgery in severe contractures.
Reality: Laser therapy improves scar texture, thickness, and minor tightness, but severe joint contractures or deep structural tissue loss require formal surgical release and tissue transfer.
Myth: Tissue expansion is suitable for every burn scar location.
Reality: Tissue expansion requires healthy adjacent skin and underlying stable bone structure. It is ideal for the scalp, neck, and trunk, but less applicable to lower limbs or small distal joints.
17. Frequently Asked Questions
What is the difference between primary burn care and secondary burn reconstruction?
Primary burn care focuses on immediate resuscitation, clearing dead tissue, infection control, and initial wound closure during the acute injury phase. Secondary burn reconstruction takes place months or years later, targeting mature scars, contractures, and functional deformities to restore mobility, comfort, and anatomical appearance.
When is the best time to undergo burn reconstruction surgery?
Reconstructive surgery is usually scheduled after the burn scar has fully matured, which takes 12 to 18 months post-injury. During this time, scars naturally soften and fade. Surgery is performed earlier only if severe contractures compromise the airway, interfere with eating, or prevent eyelid closure.
Will my skin graft match the color and texture of my surrounding skin?
Surgeons select donor sites that match the target area as closely as possible. Full-thickness grafts and expanded local tissue provide superior color and texture matches compared to thin split-thickness grafts. However, subtle differences in pigmentation and texture are normal over the long term.
How long will I need to wear custom pressure garments after surgery?
Most patients wear pressure garments for 23 hours per day for 12 to 18 months following reconstruction. Continuous pressure flattens scar tissue, controls collagen overgrowth, speeds up scar maturation, and minimizes the risk of contracture recurrence.
Is burn reconstruction covered as a medical necessity or considered cosmetic?
Burn reconstruction performed to relieve contractures, restore joint movement, correct facial distortions, or heal chronic breakdown is categorized medically as functional reconstructive surgery. Reconstructive care focuses on correcting structural defects and physical impairment rather than elective cosmetic change.
How painful is the recovery after reconstructive surgery?
Postoperative pain is managed using structured, multimodal pain management, including nerve blocks, non-opioid medications, and short-term oral narcotics. Skin graft donor sites often cause more superficial discomfort than recipient sites, but this discomfort resolves as donor skin heals over 10 to 14 days.
Can contractures return after a successful surgical release?
Contractures can recur if postoperative rehabilitation, dynamic splinting, and pressure garment therapy are omitted, or if growing children experience rapid bone growth across grafted areas. Adhering to daily therapy protocols dramatically reduces recurrence risks.
How does tissue expansion work for burn scars?
A surgeon places an inflatable silicone expander balloon beneath healthy skin next to a scar. Over several weeks, saline is added via a small port, gradually stretching the skin. Once enough skin has grown, the expander is removed, the scar excised, and the new skin stretched over the defect.
What is a microvascular free flap and when is it necessary?
A microvascular free flap involves moving skin, muscle, or fat along with its blood supply from a distant body region to a burn defect. Microsurgeons reconnect the tiny blood vessels under a microscope. Free flaps are required for deep wounds exposing bone, joint, or nerves that cannot support simple skin grafts.
How soon after surgery can I resume physical therapy and exercise?
Gentle, therapist-supervised passive range-of-motion exercises usually start between days 5 and 7 post-surgery once skin graft intake is stable. Active exercise and heavy exertion are restricted for 4 to 6 weeks to protect healing tissue structures.
How do I care for my skin graft donor site?
Donor sites are covered with specialized sterile dressings that protect exposed nerve endings and absorb fluid. Once healed (10–14 days), the new skin should be washed gently, moisturized daily with unscented lotion, and protected from sun exposure with SPF 50+ sunscreen for at least one year.
Can children undergo burn reconstruction while still growing?
Yes. Children frequently require staged reconstructive procedures as their bones grow faster than mature scar tissue can stretch. Reconstructive surgeons plan operations around developmental milestones to prevent growth restrictions and joint deformities.
What laser therapies are used alongside burn reconstruction?
Fractional carbon dioxide (CO2) lasers and pulsed-dye lasers (PDL) are frequently used alongside surgery. Laser therapy creates micro-channels in scar tissue to encourage collagen reorganization, reduce redness, smooth rough textures, and decrease chronic itching.
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